Axonal PPARγ promotes neuronal regeneration after injury

Keyphrases

• Acceptable Safety 33%
• Adipogenesis 33%
• Axonal Growth 33%
• Axonal Regeneration 33%
• Central Nerve Injury 33%
• Cortical Neurons 33%
• Drug Class 33%
• Drug Use 33%
• Dynein 33%
• Functional Role 33%
• Glucose Homeostasis 33%
• Injury Response 33%
• Molecular Motor 33%
• Molecular Targets 33%
• Motivation 33%
• Nervous System 33%
• Neural Regeneration 100%
• Neurological Disorders 33%
• Neuronal Injury 33%
• Neuronal Responses 33%
• Neuroprotection 33%
• Nuclear Accumulation 33%
• Nuclear Receptor 33%
• Peripheral Nerve Injury 33%
• Pharmaceutical Agents 33%
• PPAR Antagonist 33%
• Protein Level 33%
• Response to Injury 33%
• Retrograde Transport 33%
• Safety Profile 33%
• Sciatic Nerve Crush 33%
• Sciatic Nerve Injury 33%
• Sensory Axons 33%
• Sensory Neurons 100%
• Thiazolidinediones 33%
• Type 2 Diabetes Prevention 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Cell Nucleus Receptor 8%
• Dynein Adenosine Triphosphatase 8%
• Injury 100%
• Molecular Motor 8%
• Molecular Target 8%
• Nerve Crush 8%
• Neurologic Disease 8%
• Neuroprotection 8%
• Non Insulin Dependent Diabetes Mellitus 8%
• Peripheral Nerve Injury 8%
• Peroxisome Proliferator Activated Receptor 100%
• Peroxisome Proliferator Activated Receptor Antagonist 8%
• Sciatic Nerve Injury 8%
• Thiazolidinedione 8%